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Elucidating the specific pharmacological mechanism of action (MOA) of naturally developing compounds is often demanding. While Tarselli et al. (60) made the main de novo synthetic pathway to conolidine and showcased that this naturally occurring compound correctly suppresses responses to both equally chemically induced and inflammation-derived pain
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Conolidine’s analgesic results stem from its conversation with non-opioid pain pathways. Unlike opioids, which bind to µ-opioid receptors within the central anxious procedure, conolidine modulates alternate molecular targets. A Science Advances research discovered that conolidine interacts with the atypical chemokine receptor ACKR3/CXCR7, which